Erectile dysfunction from the fastsize
Posted: Thu Mar 08, 2012 9:08 pm
Here is my story
I am a 32 year-old male. At the age of 28, and to my eternal regret, I used a penile traction device x 8 months (~800 hours total) in an attempt to correct an asymptomatic ~15 degree congenital penile curvature/indentation after reading about it's use for Peyronie's disease in the literature. Prior to this, my IIEF-5 was consistently 25 (ie. no erectile problems whatsoever). I was not sexually active from 27.5yo to 29.9yo during busy grad school. Upon resuming sexual relations at 30, I experienced persistent moderate-to-severe ED characterized by tumescence but not rigidity or momentary rigidity followed by rapid detemuscence. I have also noticed a complete lack of spontaneous or nocturnal erections unless I am on pde-5 inhibitors. My IIEF-5 is now roughly 12. I initially attributed my ED to psychogenic ED and self-treated with pde-5 inhibitors but have come to realize recently that this is definitely not purely psychogenic. PDE-5 inhibitors work ~90% of the time with normal rigidity, but even with pde-5i I tend to detumenesce rapidly without persistent manual or coital stimulation. I am completely dependent on PDE-5 inhibitors for intercourse.
There are multiple possible mechanisms of injury from stretching - the most concerning of which is nerve damage, as this is largely not treatable except in the month to year period after injury. Stretch injury is also of a chronic nature since most people are stretching several times a week to daily, and there are no good models (eg. rat) for chronic injury in the literature. The penis is the one organ in the body which you can stretch, but there is no significant resistance from bone as in other body appendages. There really is no protection for your nerves when you are stretching your penis. Nerve stretch is one of the contributing factors to erectile dysfunction during prostatectomy.
Vascular damage is also a risk:
From http://www.the-european-institute-ugrs. ... =text#text
Patients who have used such devices have often reported that either during, or shortly after, applying them they suffered markedly impaired erections. In fact, Doppler sonography tests on them later revealed extended AA and AV shunts – in other words, bridging between blood-vessels that alter the blood-flow. Such shunts occur quite frequently and are generally congenital. However, due to long-term use of these stretching devices, which constantly compress the penile distal vessels, it would seem the shunts become distended, resulting in a permanent change in the blood-flow.
I am a 32 year-old male. At the age of 28, and to my eternal regret, I used a penile traction device x 8 months (~800 hours total) in an attempt to correct an asymptomatic ~15 degree congenital penile curvature/indentation after reading about it's use for Peyronie's disease in the literature. Prior to this, my IIEF-5 was consistently 25 (ie. no erectile problems whatsoever). I was not sexually active from 27.5yo to 29.9yo during busy grad school. Upon resuming sexual relations at 30, I experienced persistent moderate-to-severe ED characterized by tumescence but not rigidity or momentary rigidity followed by rapid detemuscence. I have also noticed a complete lack of spontaneous or nocturnal erections unless I am on pde-5 inhibitors. My IIEF-5 is now roughly 12. I initially attributed my ED to psychogenic ED and self-treated with pde-5 inhibitors but have come to realize recently that this is definitely not purely psychogenic. PDE-5 inhibitors work ~90% of the time with normal rigidity, but even with pde-5i I tend to detumenesce rapidly without persistent manual or coital stimulation. I am completely dependent on PDE-5 inhibitors for intercourse.
There are multiple possible mechanisms of injury from stretching - the most concerning of which is nerve damage, as this is largely not treatable except in the month to year period after injury. Stretch injury is also of a chronic nature since most people are stretching several times a week to daily, and there are no good models (eg. rat) for chronic injury in the literature. The penis is the one organ in the body which you can stretch, but there is no significant resistance from bone as in other body appendages. There really is no protection for your nerves when you are stretching your penis. Nerve stretch is one of the contributing factors to erectile dysfunction during prostatectomy.
Vascular damage is also a risk:
From http://www.the-european-institute-ugrs. ... =text#text
Patients who have used such devices have often reported that either during, or shortly after, applying them they suffered markedly impaired erections. In fact, Doppler sonography tests on them later revealed extended AA and AV shunts – in other words, bridging between blood-vessels that alter the blood-flow. Such shunts occur quite frequently and are generally congenital. However, due to long-term use of these stretching devices, which constantly compress the penile distal vessels, it would seem the shunts become distended, resulting in a permanent change in the blood-flow.